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monoclonal mouse anti ed 1  (Bio-Rad)


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    Bio-Rad monoclonal mouse anti ed 1
    Monoclonal Mouse Anti Ed 1, supplied by Bio-Rad, used in various techniques. Bioz Stars score: 96/100, based on 3030 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/product/mouse+monoclonal+anti+rat+ed+1/Mouse+anti+Rat+CD68/pm39602899-52-6-10
    Average 96 stars, based on 3030 article reviews
    monoclonal mouse anti ed 1 - by Bioz Stars, 2026-09
    96/100 stars

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    Related Articles

    Marker:

    Article Title: Effect of curcumin on the inflammatory reaction and functional recovery after spinal cord injury in a hyperglycemic rat model.
    Article Snippet: FDA device/ Author Disc close.. CHK: N to disclose.. KT Disclaimer: under discussi Author Con K and Y.S.L. analysis, Y.S.L https://doi.org/ 1529-9430/© 2 drug sta losures: othing K: Not None o on in th tributi ; softwa .

    Article Title: Cytokines and cell cycle regulation in the fibrous progression of crescent formation in antiglomerular basement membrane nephritis of WKY rats.
    Article Snippet: Cytokines may regulate cell proliferation by cell-cycle-regulatory proteins, in which cyclin-dependent kinase inhibitors (CDKI) inhibit cell proliferation.. We investigated whether CDKI p21 or p27, both of which are potentially regulated by transforming growth factor (TGF)-β, a key cytokine in fibrogenesis, are involved together with TGF-β and/or platelet-derived growth factor (PDGF) in the fibrous progression of glomerular crescent formation and examined the sequential change in the cell type and the cellular background of myofibroblasts in crescent formation.. Crescentic glomerulonephritis (GN) was induced by i.v. injection of rabbit antirat glomerular basement membrane antiserum in WKY rats.

    Article Title: The Neuroprotective Effect of Treatment of Valproic Acid in Acute Spinal Cord Injury
    Article Snippet: .. To detect ED-1 (marker for activated macrophages) and histone acetylation, spinal cord sections were blocked with 4% normal serum in 0.5% Triton X-100 for 1 hour at room temperature and incubated overnight at 4°C with a 1 : 500 dilution of mouse monoclonal anti-rat ED-1 (Serotec, Oxford, UK) and a 1 : 1000 dilution of polyclonal anti-rat acetyl-histone H3 (K9, Ac-H3/K9; Cell Signaling Technology, Danvers, MA, USA) and a 1 : 1000 dilution of polyclonal anti-rat acetyl-histone H3 (K18, Ac-H3/K18; Cell Signaling Technology), and rinsed for 3×10 min in 0.1 M PB. ..

    Incubation:

    Article Title: Effect of curcumin on the inflammatory reaction and functional recovery after spinal cord injury in a hyperglycemic rat model.
    Article Snippet: FDA device/ Author Disc close.. CHK: N to disclose.. KT Disclaimer: under discussi Author Con K and Y.S.L. analysis, Y.S.L https://doi.org/ 1529-9430/© 2 drug sta losures: othing K: Not None o on in th tributi ; softwa .

    Article Title: Cytokines and cell cycle regulation in the fibrous progression of crescent formation in antiglomerular basement membrane nephritis of WKY rats.
    Article Snippet: Cytokines may regulate cell proliferation by cell-cycle-regulatory proteins, in which cyclin-dependent kinase inhibitors (CDKI) inhibit cell proliferation.. We investigated whether CDKI p21 or p27, both of which are potentially regulated by transforming growth factor (TGF)-β, a key cytokine in fibrogenesis, are involved together with TGF-β and/or platelet-derived growth factor (PDGF) in the fibrous progression of glomerular crescent formation and examined the sequential change in the cell type and the cellular background of myofibroblasts in crescent formation.. Crescentic glomerulonephritis (GN) was induced by i.v. injection of rabbit antirat glomerular basement membrane antiserum in WKY rats.

    Article Title: The Neuroprotective Effect of Treatment of Valproic Acid in Acute Spinal Cord Injury
    Article Snippet: .. To detect ED-1 (marker for activated macrophages) and histone acetylation, spinal cord sections were blocked with 4% normal serum in 0.5% Triton X-100 for 1 hour at room temperature and incubated overnight at 4°C with a 1 : 500 dilution of mouse monoclonal anti-rat ED-1 (Serotec, Oxford, UK) and a 1 : 1000 dilution of polyclonal anti-rat acetyl-histone H3 (K9, Ac-H3/K9; Cell Signaling Technology, Danvers, MA, USA) and a 1 : 1000 dilution of polyclonal anti-rat acetyl-histone H3 (K18, Ac-H3/K18; Cell Signaling Technology), and rinsed for 3×10 min in 0.1 M PB. ..



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    a Experimental scheme depicting the induction of anti-GBM nephritis followed by intravenous ASC and BMMSC administration on days 0, 2, and 4. b Serum creatinine level on days 3 and 7 (day 3, n = 5 per group; day 7, n = 8 per group). c Urinary protein excretion on day 3 ( n = 8 per group). d Representative images of kidney sections stained with Periodic Acid-Schiff on day 7 (left) and glomerular injury scoring based on crescent formation on days 3 and 7 (right). e Representative images of kidney sections stained with <t>anti-CD68</t> Ab on day 7 (left) and the number of <t>CD68-positive</t> cells in the glomerulus on days 3 and 7 (right). Scale bars, 50 μm. (day 3, n = 5 per group; day 7, n = 8–9 per group). All data are shown as means ± SD. * p < 0.05, ** p < 0.01, *** p < 0.001, as determined by ANOVA. ASC adipose-derived MSC, BMMSC bone marrow-derived MSC.
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    a Experimental scheme depicting the induction of anti-GBM nephritis followed by intravenous ASC and BMMSC administration on days 0, 2, and 4. b Serum creatinine level on days 3 and 7 (day 3, n = 5 per group; day 7, n = 8 per group). c Urinary protein excretion on day 3 ( n = 8 per group). d Representative images of kidney sections stained with Periodic Acid-Schiff on day 7 (left) and glomerular injury scoring based on crescent formation on days 3 and 7 (right). e Representative images of kidney sections stained with <t>anti-CD68</t> Ab on day 7 (left) and the number of <t>CD68-positive</t> cells in the glomerulus on days 3 and 7 (right). Scale bars, 50 μm. (day 3, n = 5 per group; day 7, n = 8–9 per group). All data are shown as means ± SD. * p < 0.05, ** p < 0.01, *** p < 0.001, as determined by ANOVA. ASC adipose-derived MSC, BMMSC bone marrow-derived MSC.
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    a Experimental scheme depicting the induction of anti-GBM nephritis followed by intravenous ASC and BMMSC administration on days 0, 2, and 4. b Serum creatinine level on days 3 and 7 (day 3, n = 5 per group; day 7, n = 8 per group). c Urinary protein excretion on day 3 ( n = 8 per group). d Representative images of kidney sections stained with Periodic Acid-Schiff on day 7 (left) and glomerular injury scoring based on crescent formation on days 3 and 7 (right). e Representative images of kidney sections stained with <t>anti-CD68</t> Ab on day 7 (left) and the number of <t>CD68-positive</t> cells in the glomerulus on days 3 and 7 (right). Scale bars, 50 μm. (day 3, n = 5 per group; day 7, n = 8–9 per group). All data are shown as means ± SD. * p < 0.05, ** p < 0.01, *** p < 0.001, as determined by ANOVA. ASC adipose-derived MSC, BMMSC bone marrow-derived MSC.
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    a Experimental scheme depicting the induction of anti-GBM nephritis followed by intravenous ASC and BMMSC administration on days 0, 2, and 4. b Serum creatinine level on days 3 and 7 (day 3, n = 5 per group; day 7, n = 8 per group). c Urinary protein excretion on day 3 ( n = 8 per group). d Representative images of kidney sections stained with Periodic Acid-Schiff on day 7 (left) and glomerular injury scoring based on crescent formation on days 3 and 7 (right). e Representative images of kidney sections stained with <t>anti-CD68</t> Ab on day 7 (left) and the number of <t>CD68-positive</t> cells in the glomerulus on days 3 and 7 (right). Scale bars, 50 μm. (day 3, n = 5 per group; day 7, n = 8–9 per group). All data are shown as means ± SD. * p < 0.05, ** p < 0.01, *** p < 0.001, as determined by ANOVA. ASC adipose-derived MSC, BMMSC bone marrow-derived MSC.
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    a Experimental scheme depicting the induction of anti-GBM nephritis followed by intravenous ASC and BMMSC administration on days 0, 2, and 4. b Serum creatinine level on days 3 and 7 (day 3, n = 5 per group; day 7, n = 8 per group). c Urinary protein excretion on day 3 ( n = 8 per group). d Representative images of kidney sections stained with Periodic Acid-Schiff on day 7 (left) and glomerular injury scoring based on crescent formation on days 3 and 7 (right). e Representative images of kidney sections stained with <t>anti-CD68</t> Ab on day 7 (left) and the number of <t>CD68-positive</t> cells in the glomerulus on days 3 and 7 (right). Scale bars, 50 μm. (day 3, n = 5 per group; day 7, n = 8–9 per group). All data are shown as means ± SD. * p < 0.05, ** p < 0.01, *** p < 0.001, as determined by ANOVA. ASC adipose-derived MSC, BMMSC bone marrow-derived MSC.
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    Image Search Results


    a Experimental scheme depicting the induction of anti-GBM nephritis followed by intravenous ASC and BMMSC administration on days 0, 2, and 4. b Serum creatinine level on days 3 and 7 (day 3, n = 5 per group; day 7, n = 8 per group). c Urinary protein excretion on day 3 ( n = 8 per group). d Representative images of kidney sections stained with Periodic Acid-Schiff on day 7 (left) and glomerular injury scoring based on crescent formation on days 3 and 7 (right). e Representative images of kidney sections stained with anti-CD68 Ab on day 7 (left) and the number of CD68-positive cells in the glomerulus on days 3 and 7 (right). Scale bars, 50 μm. (day 3, n = 5 per group; day 7, n = 8–9 per group). All data are shown as means ± SD. * p < 0.05, ** p < 0.01, *** p < 0.001, as determined by ANOVA. ASC adipose-derived MSC, BMMSC bone marrow-derived MSC.

    Journal: Communications Biology

    Article Title: Mesenchymal stem cells exert renoprotection via extracellular vesicle-mediated modulation of M2 macrophages and spleen-kidney network

    doi: 10.1038/s42003-022-03712-2

    Figure Lengend Snippet: a Experimental scheme depicting the induction of anti-GBM nephritis followed by intravenous ASC and BMMSC administration on days 0, 2, and 4. b Serum creatinine level on days 3 and 7 (day 3, n = 5 per group; day 7, n = 8 per group). c Urinary protein excretion on day 3 ( n = 8 per group). d Representative images of kidney sections stained with Periodic Acid-Schiff on day 7 (left) and glomerular injury scoring based on crescent formation on days 3 and 7 (right). e Representative images of kidney sections stained with anti-CD68 Ab on day 7 (left) and the number of CD68-positive cells in the glomerulus on days 3 and 7 (right). Scale bars, 50 μm. (day 3, n = 5 per group; day 7, n = 8–9 per group). All data are shown as means ± SD. * p < 0.05, ** p < 0.01, *** p < 0.001, as determined by ANOVA. ASC adipose-derived MSC, BMMSC bone marrow-derived MSC.

    Article Snippet: To evaluate macrophage infiltration, formalin-fixed kidneys were stained using mouse anti-rat CD68 monoclonal IgG1 (clone ED-1; BMA Biomedicals, Augst, Switzerland) as a marker for infiltrating macrophages or dendritic cells.

    Techniques: Staining, Derivative Assay